Scientists have identified a critical function of what they believe to be schizophrenia's from a very large caseidf "Rosetta Stone" gene that could hold the key to decoding the function of all genes can involved in the disease dentified a critical function of what they believe to be schiz. am, mattis pharetra aliquet sed, venenatis quis neque. Pellentesque pellentesque iaculis porta. Nam quis odio vitae lectus convallis accumsan et sed neque. Integer eget mi eget risus eleifend consectetur vehicula sit amet neque.

Breakthrough reveals gene's influence in a vulnerable period of the brain's development!

The breakthrough has revealed a vulnerable period in the early stages of the brain's development that researchers hope can be targeted for future efforts in reversing schizophrenia. The breakthrough has revealed a vulnerable period in the early stages of the brain's development that researchers hope can be targeted for future efforts in reversing schizophrenia.

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Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain's largest region from being able to form synapses.

We believe that DISC-1 is schizophrenia's Rosetta Stone gene and could hold the master key to help us unlock our understand of the role played by all risk genes involved in the disease.

Prof. John Doe , Chaos University.

Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain's largest region from being able to form synapses.

Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain's largest region from being able to form synapses.

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Description of the image and the referece of it will be here.

Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments in mice.

Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments in mice.

image hover
Description of the image and the referece of it will be here.

Evealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain. Their experiments in mice revealed that by preventing DISC-1 from binding with these molecules - using a protein-releasing drug called Tamoxifen at an early stage of the brain's development - it would lack plasticity once it grows to its adult state, preventing cells (cortical neurons) in the brain.